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  • 121 -2 (30/3) 2020 — Negmatshaeva H. N., — PERINATAL AND HEMODYNAMIC EVALUATION OF THE USE OF OZONE THERAPY IN THE TREATMENT OF PREECLAMPSIA

121 -2 (30/3) 2020 — Negmatshaeva H. N., — PERINATAL AND HEMODYNAMIC EVALUATION OF THE USE OF OZONE THERAPY IN THE TREATMENT OF PREECLAMPSIA

PERINATAL AND HEMODYNAMIC EVALUATION OF THE USE OF OZONE THERAPY IN THE TREATMENT OF PREECLAMPSIA

Negmatshaeva H. N.,- Andijan State Medical Institute, 110000 Uzbekistan, Andijan Navoii avenyu 126 http://adti.uz.

Resume,

Preeclampsia, being a multi-organ pathology, in modern obstetrics to this day remains an urgent problem, causing the interest of world researchers. There is no consensus on the prevention of this pathology and the solution of its further complications.

Keywords: preeclampsia, pregnancy, obstetric complications, prevention

First page

462

Last page

464

For citation: Negmatshaeva H. N., Perinatal and hemodynamic evaluation of the use of ozone therapy in the treatment of preeclampsia//New Day in Medicine 2(30)2020 462-464 https://cutt.ly/gvIH9vO

List of References

  1. Анализ причин материнской смертности: Руководство для врачей. Под ред. проф. А.П. Милованова. М: МДВ 2008. С. 228.
  2. Ахмедов Ф.К. Изучение роли почечного кровотока и концен­трации мочевой кислоты в крови и моче в диагностике пре­эклампсии // Журнал теоретической и клинической медици­ны. – Ташкент, 2015, № 3. – С. 63-66.
  3. Милованов А.П., Кириченко А.К. Цитотрофобластическая инвазия – ключевой механизм развития нормальной и ослож­ненной беременности. Красноярск 2009. С. 211.
  4. Курбонова З.Ш., Ахмедов Ф.К. Мочевая кислота – маркер развития преэклапсия // Новости дерматологии и репродук­тивного здоровья – Ташкент, 2017, № 3-4 (II) – С. 27-29.
  5. Akhmedov F.K. Peculiarities of cardiac hemodynamic in pregnant women with mild preeclampsia // Europen Science Review. – Austria, Vienna, 2015, № 4-5 – С. 56-58.
  6. Brosens 1.А., Robertson W.B., Dixon H.G. The physiological response of the vessels of the placental bed to normal pregnancy. J Pathol Bacteriol 1967; 93: 569-579.
  7. Burger D., Touyz R.M. Cellular biomarkers of endothelial health: microparticles, endothelial progenitor cells, and circulating endothelial cells. Am Soc Hypertension 2012; 6: 2: 85-99?
  8. Lamarca B. Endothelial dysfunction. An important mediator in the pathophysiology of hypertension during pre-eclampsia. Minerva Ginecol 2012; 64: 4: 309-320.
  9. Marusic J., Prusac I.K., Tomas S.Z., Karara J.R., Roje D. Expression of inflammatory cytokines in placentas from pregnancies complicated with preeclampsia and HELLP syndrome. J Matern Fetal Neonatal Med 2013; 26: 7: 680-685.
  10. Meekins J.W., Pijnenborg R., Hanssens M., McFadyen I.R., van Asshe A. A study of placental bed spiral arteries and trophoblast invasion in normal and severe pre-eclamptic pregnancies. Br J Obstet Gynaecol 1994; 101: 669-674.
  11. Wang A., Rana S., and Karumanchi S.A. Preeclampsia: the role of angiogenic factors in its pathogenesis. Physiology (Bethesda) 2009; 24: 147-158.
  12. Dekker G., Robillard P.Y., Roberts C. The etiology of preeclampsia: the role of the father. J Reprod Immunol 2011; 89: 2: 126-32.

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